【手腕痛到喊】任何人都可以患上「媽媽手」
⭐做家務搬運重物要小心
⭐過度打機滑手機都高危
#星期二提升正能量
手腕痛?三秒測試「媽媽手」
初生嬰兒需要多抱抱,令不少媽媽慘痛地患上「媽媽手」,手腕疼痛,不能彎曲及用力,痛到連毛巾都扭不到。「媽媽手」其實是橈骨莖突狹窄性腱鞘炎 ,是因為經常持久地外展拇指 , 導致手腕肌腱在不斷增厚而又狹窄的腱鞘內不斷地運動摩擦以致產生炎症。想知道你的手腕痛是否「媽媽手」,可以進行以下測試:
「媽媽手」測試
方法:指尖向前手掌打開,拇指貼近掌心,用餘下4隻手指包着拇指,將手腕向下拉。
感到拉扯感 → 正常
感到疼痛 → 你可能患上媽媽手了
如果患上「媽媽手」,必須讓手休息,減少拇指用力的機會,有時候抱嬰兒的動作可以反過來,以手背支撐,另一隻手加護,不要長時間以手掌朝天抱嬰兒 。平日用暖毛巾熱敷患處約10分鐘,做一些手部拉筋運動。 從中醫角度來看,患肌腱炎時不宜食用糯米、牛奶、南瓜、大豆等生痰及滯氣發物,因為容易阻塞經絡;亦要減少食用冷、酸、辣、油膩等食物。
紓緩「媽媽手」手指運動:
手腕背伸,五指張開、伸直朝天,維持10秒,重覆動作10次。
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Wrist pain? A quick test for ‘Mommy Thumb’
Newborns need to be carried all the time. This is how mothers develop ‘mommy thumb’, or in medical terms, De Quervain's disease. The sufferers would experience wrist pain, and they could not bend their wrists or exert pressure on their hands. Some might not even be able to wring a towel.
De Quervain's disease is actually stenosing tenosynovitis of the styloid process of radius and due to regular thumb abduction. As the muscles and tendons on the wrists thicken, the friction become more intense, causing inflammation in the hands. In order to find out if the wrist pain you experience is due to De Quervain's disease, you can carry out the following test:
The ‘mommy thumb’ test
Method: stretch your palms and make sure your fingers point outwards. Stick your thumb close to the palm, and use the remaining fingers to wrap around the thumb, and bend your wrist downwards. It is normal if you feel something is stretching from within, but if you feel pain during the movement, you might have a mommy thumb.
If you do get a mommy thumb, make sure you give your hands enough rest and avoid exerting pressure on your thumb. You can carry the baby the other way round, by supporting the baby with the back of one hand, and hold him or her up with the other. However, avoid carrying the baby with the back of your hand for an extended period.
When we experience inflammation in the wrist (symptoms include redness, swelling, warm, and painful), apply cold compress with a towel for about 10 minutes. On regular days, apply warm compress on the same area for 10 minutes and do some hand and wrist stretches. From the perspective of Chinese Medicine, an individual who suffers from tendinitis should avoid taking glutinous rice, milk, pumpkin, and soybean as these ingredients induce phlegm and stagnate qi, and would impede the circulation of the meridians. Cut down on raw, cold, sour, spicy, and greasy foods as well.
Finger exercise to relieve ‘mommy thumb’
Point the fingers skyward and stretch the palm. Curl the thumb and little finger inward so they meet at the center of the palm (as if we are making a vow). Then stretch the thumb towards the little finger for 10 seconds. Repeat the process 10 times.
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同時也有3部Youtube影片,追蹤數超過5,660的網紅利惟庸Tony Li,也在其Youtube影片中提到,Thanks to Jason and the one who ask me play this , I ‘ve got Tendinitis after doing this shit . Thanks ! Thanks a lot , REALLY ! I 'm not good at shr...
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tendinitis 在 健康世界 Facebook 的精選貼文
小心負重太重 #肌腱炎(Tendinitis)發作!
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【踮起腳尖痛,腳踝也會有夾擠問題?】
(這次文章內容稍長,若懶得看文字內容可直接觀看影片)
大家應該對於肩夾擠、髖夾擠這兩個名詞不陌生,但你有聽過腳踝夾擠嗎?夾擠指的是我們的骨頭過度擠壓到周遭的軟組織,可能是肌腱、韌帶或是滑液膜等等,造成疼痛或角度受限。夾擠是一個症候群,並非一個特定的病症,夾擠症候群底這個名詞底下,可能夾到的組織不同,造成的原因歧異度也非常大,造成評估上其實並不是那麼容易。腳踝夾擠雖然沒有像肩夾擠一樣有被正式分類成不同的夾擠類型,但仍能根據症狀呈現的方式跟解剖構造簡單分為前夾擠跟後夾擠,若還要再細分還會分前內側、前外側夾擠,以及後內側、後外側夾擠。
前側夾擠的症狀主要出現在腳踝背屈末端角度的時候,脛距關節 (Tibiotalar joint) 前側的組織受到擠壓。腳踝前側的有不少脂肪、滑囊組織,正常情況,這些組織會在腳踝背屈15度過後受到擠壓,但如果在遠端脛骨前側或是距骨頸有增生的骨頭的話,便可能限縮前側的空間,讓組織提早受到壓迫。如果長期在這角度下活動,就可能進一步造成慢性的發炎,或是造成關節囊韌帶的增生。除此之外,如果腳踝曾經扭過傷,造成韌帶或皺襞增厚的話,也是可能造成前側夾擠的原因之一。
雖然這些解剖構造上的變化已有多篇文章有所描述。但造成這些組織增生的原因卻仍不是很清楚。因為運動員有比較高的比例有這樣的問題,有些學者認為前側夾擠可能是因為頻繁地做出大角度的背屈,或是因為運動過程中受到的外力,讓前側軟骨邊緣反覆受到衝擊所造成。也有些學者認為,踝關節的不穩定,造成關節有不正常的微小滑動,也是一個可能造成骨質增生、或是軟組織受到夾擠的的原因。另外在比較早期的文章,一開始學者認為前側的骨質增生可能是來自於頻繁地蹠屈,牽拉到關節囊,進而造成關節處的增生,只是這樣的假設被後來的研究給推翻了。
因為前側夾擠症狀大多是在腳踝背屈的末端角度下出現,上樓梯、跑步、走上坡、爬梯還有深蹲是幾個比較容易會加劇前側疼痛的活動。若未接受妥善治療,在症狀後期可能會因為組織的增生或疼痛,造成更進一步的活動度受限、夾擠和周圍組織的傷害,再回頭限制關節活動度與功能,形成惡性循環。
後側夾擠的症狀主要出現在腳踝蹠屈到末端角度的時候,脛距關節與距跟關節後側的組織受到擠壓。後側夾擠常出現在需要頻繁把腳踝往下壓的人身上,像是芭蕾舞者、需要頻繁跳躍的運動員等等。與前側夾擠雷同,後側夾擠可能是骨質或是軟組織的夾擠,或是兩者同時存在。距骨後外側 (trigonal process) 的骨質增生是比較常被認為導致後側夾擠的原因。除此之外,頻繁的將腳板大幅度的往下踩,可能會導致後側關節囊、後下脛腓韌帶、三角韌帶的後側韌帶發炎,產生疤痕組織,進而造成組織增厚。另外我們的屈足拇長肌的肌腱經過距骨後側的內、外骨突中間的凹槽,也很容易因為過度使用,或是周遭骨質的增生,造成肌腱病變,像是肌腱或腱鞘炎的問題。
與前側夾擠的疼痛大多較為淺層、可觸摸的到相反,後側夾擠的症狀通常較為模糊,比較難有一個特定的單點疼痛,而且位置較深,通常落在阿基里斯腱底下。這也讓後側夾擠不容易和阿基里斯腱或是腓骨長肌的問題做區分。因為症狀出現在腳踝往下踩的時候,走下坡、下樓梯或是穿鞋跟較高的鞋子是幾個容易誘發症狀出現的活動。芭蕾舞者之所以比較容易出現這樣的症狀,被認為是因為需要頻繁的做出踮腳站,承重在前足的關係。
雖然影像檢查出來的骨質、軟組織的病變被認為是可能導致腳踝夾擠的原因之一,但實際上研究還是有提到,我們仍然不能光靠這些影像結果證據就判斷踝關節是否夾擠。影像檢查與我們的症狀表現之間的相關程度有限,仍需要結合其他理學檢查做綜合判斷才行。針對踝關節夾擠的介入,目前比較常見的作法仍是先採取保守治療,若在急性疼痛期,需要先避免會造成疼痛的動作,有必要的話也會使用消炎藥來控制疼痛。在非急性期,甚至是已經是慢性問題的個案,我們則需要著重在踝關節穩定、本體感覺的訓練上,畢竟前面有提到,踝關節不穩、扭傷都是可能造成夾擠的原因之一。與其他肌肉骨骼問題一樣,即使解剖構造上的異常也會被認為是造成踝關節夾擠的原因,但大多數的個案都能在不開刀的情況下有很好的進步。若有類似的狀況,一樣記得先找醫療人員的協助,避免症狀隨著時間越變越嚴重。底下的影片 (6:52) 將跟大家分享幾個簡單的踝關節穩定與本體感覺的訓練。
Impingement syndrome is a common musculoskeletal problem in shoulder and hip joints. But have you ever heard of ankle impingement? Impingement syndrome refers to abnormal contact of bony structures or soft tissue, e.g., tendon, ligament, synovial tissue, resulting in pain and restriction. Through different causes of impingement syndrome, it includes different medical signs or symptoms. Therefore, causes of impingement syndrome differ from person to person, making it more difficult to make a right diagnosis. Although ankle impingement is not officially classified into different types like shoulder impingement, researchers still sort it into anterior and posterior impingement according to anatomical structures are involved. More specifically, it can be classified into anteriomedial, anteriolateral, posteriomedial and posteriolateral impingement.
Symptoms of anterior ankle impingement are generally induced by compression of anterior margin of tibiotalar joint in terminal dorsiflexion. There are adipose and synovial tissues in the anterior joint space. Normally, these tissues are compressed after 15 degree of dorsiflexion in healthy individuals. However, if there is osteophyte at anterior distal tibia or talus neck, it will take up the space and limit ankle movement, causing early compression. This will result in chronic inflammation, synovitis, and capsuloligamentous hypertrophy. Apart from this, ankle sprain, thickened anterior tibiofibular ligament and synovial plica are also possible causative factors.
Even though structural pathologies are well described in much research, their exact etiologies are still less understood. Research showed that athletes are tend to affected by anterior impingement, and it led to hypothesis that pathologies are caused by repetitive impact injury to anterior chondral margin from hyper-dorsiflexion or direct impact during sports. Chronic ankle instability has also been hypothesized to be the causative factor of anterior impingement, because abnormal repetitive micromotion may develop bony and soft tissue lesions. In addition, early research hypothesized anterior osteophyte is caused by traction to the anterior capsule during repetitive plantar flexion, but this theory was disproved by later anatomic studies.
Anterior impingement symptom typically presents as anterior ankle pain during terminal dorsiflexion. Climbing stairs, running, walking up hills, ascending ladders and deep squat are common aggravating activities. If anterior impingement doesn’t get treated well, in the later stage, joint mobility may be further restricted due to mechanical block or pain, resulting in vicious circle.
Posterior ankle impingement symptom typically occurs in terminal plantarflexion, due to compression of tissues posterior to the tibiotalar and talocalcaneal joint. Posterior impingement tend to occur in athletes who need to plantarflex frequently, like ballet dancers, etc. Similarly, posterior impingement can result from compression of bony or soft tissue in isolation or in combination. Trigonal process of posterior talus is the most common cause of posterior impingement. Besides this, repetitive hyper-plantarflexion may cause posterior capsule, inferior tibiofibular ligament, and posterior fiber of deltoid ligament inflammation, scarring, and thickening. Lastly, tendinitis and tenosynovitis are easily found in flexor hallucis longus tendon, running between the medial and lateral posterior process of the talus. This probably results from overuse or irritation from surrounding abnormal bony tissue. The tissues mentioned above are all possible causative factors to the posterior ankle impingement.
In contrast to patients with anterior impingement pain that are accessible to palpation, posterior impingement pain is less specific, deep to the Achilles tendon. This makes it difficult to differentiate from Achilles tendon or peroneal tendon pathology. Since posterior impingement symptom is usually irritated by repetitive plantarflexion, walking downstairs, downhill running, and wearing high-heeled shoes are some common exacerbated activities to posterior impingement syndrome. Ballet dancers are commonly affected by posterior impingement syndrome due to weight bearing on forefoot in plantarflexion position over and over again.
Though osseous or soft tissues abnormality in radiography is seen to be one of the causes of ankle impingement, it doesn’t mean that we can simply blame patient’s symptom on these structural pathology. In fact, there is a limited correlation between medical image findings and our symptom. We should integrate patient’s history, physical examination, imaging studies, etc., for accurate diagnosis. Conservative treatment remains first option to manage ankle impingement. In acute stage, patient should avoid from doing provocative activities. If it is necessary, NSAIDs can be used for pain management. In chronic stage, clinicians should focus on ankle stability and proprioception training because ankle instability and sprain are both causative factors of ankle impingement. Just like other musculoskeletal disease, even though structural abnormality is thought to be a possible cause of ankle impingement, most ankle impingement cases still respond well to conservative treatment. If you have any similar medical problem, please find medical professions for help. The video below will show you some simple ways to train our ankle stability and proprioception.
參考資料:
https://pubmed.ncbi.nlm.nih.gov/27608626/
https://link.springer.com/article/10.1007/s00247-019-04459-5
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5065672/
#腳踝夾擠 #踝關節不穩 #腳踝扭傷 #本體感覺訓練 #物理治療 #ankleimingement #ankleinstability #anklesprain #proprioception #physiotherapy #hunterptworkout
tendinitis 在 利惟庸Tony Li Youtube 的精選貼文
Thanks to Jason and the one who ask me play this , I ‘ve got Tendinitis after doing this shit . Thanks ! Thanks a lot , REALLY !
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tendinitis 在 pennyccw Youtube 的最佳解答
For one night Elton Brand enjoyed a reprieve.
For one night Brand left behind all the losses in rebuilding and tasted what it's like to be a champion.
Of course he had to go to Minneapolis on Monday night and watch his college team to do it. Reality returned quickly, not to mention embarrassingly.
Brand's experience watching Duke's national championship victory over Arizona contrasted sharply with what he experienced 24 hours later at the United Center.
That's when an announced sellout crowd of 21,749 spent the entire fourth quarter cheering for Detroit guard Jerry Stackhouse to top 50 points en route to a record-breaking 57 points in a 110-83 Pistons blowout.
When Stackhouse stylishly cracked 50 on a breakaway reverse dunk with 2 minutes 41 seconds remaining, those who remained in the stands cheered loudly. Then the action deteriorated into a playground game with Pistons passing up open shots--some of them on breakaways--to set up Stackhouse. Fans booed A.J. Guyton as he dribbled out the clock, depriving Stackhouse of one more shot.
Stackhouse still did plenty of damage, finishing with an NBA-season-high and Pistons franchise-record 57. He also eclipsed Michael Jordan's United Center record of 53 points, set against Detroit on March 7, 1996, and tied a building record with 21 field goals.
"Guys wanted the record more than I did," Stackhouse said. "They made the extra effort and that typifies what this team is about."
That effort dominated coach Tim Floyd's postgame comments--or, more accurately, the lack thereof. Floyd angrily ripped his team, rendering only Brand and Fred Hoiberg immune.
"It was a pathetic effort," Floyd said. "Our fans deserved better than that."
Those fans gave Stackhouse a standing ovation as he exited with 6.9 seconds remaining.
"If our fans were enjoying it based on everything else they saw from our club that they paid to come watch, then so be it," Floyd said. "I do have a problem, not from their end, but from our end with that guy going to the rim every time he wanted to go to the rim. I had a heckuva problem with that."
Floyd might have even a bigger problem with Ron Artest's assessment of Stackhouse's game.
"It was cool watching it from the bench," said Artest, one of the many defenders Stackhouse victimized.
The Bulls have lost 13 of their last 14 and were swept in a season series by the Pistons for just the fourth time in franchise history. Their average margin of defeat was 19.8 points.
Here's all you need to know: After one quarter the scoreboard read Stackhouse 24, Bulls 20.
The first-quarter performance tied Dominique Wilkins' mark set Jan. 29, 1988, for most points in one quarter against the Bulls. It also broke Scottie Pippen's 1997 record of 21 for most points scored in one quarter at the United Center and tied Isiah Thomas' and Joe Dumars' Pistons franchise mark for most points in a quarter.
Stackhouse's night marked the eighth time he has topped 40 points this season. His previous high was 46.
Jamal Crawford tied his career high with 17 points to lead the Bulls, who played without Ron Mercer. The guard sat with tendinitis in his right ankle.
STACKING UP RECORDS
Jerry Stackhouse's 57-point performance Tuesday night set several records:
- Pistons' single-game scoring mark: Old mark, 56, Kelly Tripucka, 1-29-83 vs. Bulls.
- United Center scoring mark: Old mark, 53, Michael Jordan, 3-27-96 vs. Detroit.
- United Center field goals made mark: 21, tied Jordan vs. Detroit, 3-27-96.
- Pistons' one-quarter scoring mark: 24 in the first quarter tied Isiah Thomas (three times) and Joe Dumars (once). He outscored the Bulls 24-20 in the first.
tendinitis 在 pennyccw Youtube 的最讚貼文
Now that they are finally healthy, the Golden State Warriors think they can make a run at a playoff spot.
Baron Davis scored 22 points, and the Warriors took advantage of Carmelo Anthony's absence to beat the Denver Nuggets 110-96 on Wednesday night.
All five starters scored in double figures for the Warriors (28-35), who overcame a 12-point second-quarter deficit and took control of the game with a 14-4 outburst to start the third quarter. The Warriors were 16-of-34 from 3-point range, including four each by Jason Richardson and Stephen Jackson.
"They've all had their shots at us when haven't been together," Warriors coach Don Nelson said. "It's been a long time getting here, but I like the way the team's playing. Everyone's fired up to have a full squad."
Davis, who has missed 18 games, had nine assists and five rebounds. Jason Richardson, who missed 31 games, had 16 points, six rebounds and four assists.
But Nelson singled out recent addition Jackson, who has only been with the team for 19 games after coming over in a trade with Indiana, as the key to this win. Jackson had 18 points and eight assists, despite being hampered by tendinitis in his left knee and a troublesome left big toe.
"Jackson did a really good job in the second half on Allen Iverson," Nelson said. "I didn't even know if I'd have him in the second half. Once I put him in, I wasn't going to take him out because I knew he'd stiffen up."
Al Harrington, who scored 20 points to go along with nine rebounds, added three 3-pointers.
"They can really shoot the ball," Nuggets coach George Karl said. "We gave them their strength way too often with the corner 3, which they like to shoot."
Iverson led Denver (29-30) with 35 points, but only surprise starter DerMarr Johnson joined him in double figures with 15. Anthony, the NBA's leading scorer, remained in Denver after the birth of his first child.
"I thought we had a great game going until about the four-minute mark of the second quarter," Karl said. "They got back into the game, and in the second half, we never got into an offensive rhythm. We never got A.I. into the game like we did in the first half."
Iverson had only 11 points after halftime.
"I didn't finish like I did in the first half," Iverson said. "I just struggled for whatever reason, at the wrong time of the game."
Iverson helped the Nuggets open a 48-36 lead with 3:54 remaining in the second quarter. The Warriors used the 3-pointer to get back in the game, making four in a 16-3 run that gave them a 54-53 lead.
Davis made three free throws after being fouled on a desperation 3-pointer with 0.6 seconds left in the half, giving Golden State a 59-58 lead.
Iverson cooled in the third quarter, missing his first five shots, and the Denver offense disappeared along with his. The Nuggets were just 4-of-23 in the period while the Warriors seized control.
Golden State stretched its lead to 93-73 midway through the fourth quarter. The Nuggets cut it to 98-89 with 3:08 left, but got no closer.
The Warriors, plagued by injuries all season, are suddenly feeling confident.
"The last three games, we've been playing together, playing as a team," Richardson said. "We're playing at a high level right now."
Game notes
Anthony missed the game to be with his fiancee, MTV's LaLa Vazquez, who had the couple's first child earlier Wednesday. Kiyan (pronounced Kai-ann) Anthony arrived weighing 5 lbs., 9 oz. and is 18 1/2 inches long. Warriors coach Don Nelson said the Warriors are "still in the hunt," despite his statement last week that the team wasn't going to get to postseason. Nelson said that quote was taken out of context. Johnson didn't get off the bench against New Orleans on Tuesday night. He finished with 12 points in 30 minutes Wednesday.
tendinitis 在 Innovative Treatment of Tendinitis - YouTube 的推薦與評價
Most people develop tendinitis because their jobs or hobbies involve repetitive motions, which put stress on the tendons needed to perform ... ... <看更多>